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Yin Y, Morgunova E, Jolma A, Kaasinen E, Sahu B, Khund-Sayeed S, et al

46,47 These effects are mediated by SIRT1 pathway activation, which optimizes mitochondrial function and autophagy, and by miRNA-dependent regulation (e.g., miR-146a targeting Nrf2/NF-B pathways), although the mechanisms vary across cell types and microenvironments

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found in their study that in clear cell renal cell carcinoma (ccRCC) tumor cells, endogenous Gln metabolism might reduce the anti-tumor effectiveness of T cells by promoting the Treg cell response (41)

We further show that elevated glycolytic capacity in CD1c hi DC is associated with increased abundance of GLUT1, MCT1 and PFKFB4, and elevated PDK1, which skews glucose homeostasis 35 by preventing pyruvate shuttling into TCA into mitochondria