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Metabolic adaptations to acute glucose uptake inhibition converge upon mitochondrial respiration for leukemia cell survival

Glioblastoma at the crossroads: current understanding and future therapeutic horizons

navitoclax (ABT-263) and ABT-737 inhibit the anti-apoptotic protein family Bcl2/BCL-xL, consequently triggering mitochondrial-mediated apoptosis [8, 9], and the cardiac glycosides ouabain has a similar effect

Under iron stimulation, HFE-deficient Kupffer cells phagocytose LDL-C in large amounts and reduce blood lipid levels ( The discovery of ferroptosis may explain the biological effects of iron accumulation

Mice were further housed individually one week before conducting social behavior assays