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Beyond the acute phase, improper adaptive immune responses contribute to LC, characterized by systemic inflammation, immune dysregulation, and abnormal CD4 and CD8 T-cell behavior (13, 14)

Other investigators have attempted to facilitate the availability of central brain-derived neurotrophic factor (BDNF) with injections of human BDNF directly into the patients brain with minimal positive results [17]

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Glial sodium dependent transporters, GLAST (EAAT1) and GLT1 (EAAT2), in particular GLT1, are responsible for at least 90% of extracellular glutamate removal (For review, see Anderson and Swanson, 2006)