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Rapamycin was shown in a mouse model of Parkinsons to preserve glutamate transporter function, keeping glutamate at the right levels.[ref] Glutamate receptors: The availability of glutamate receptors is the other half of the picture for both stimulation of the neurons and good brain function as well as for removing glutamate from the synapse
In cerebral ischemia-reperfusion models, riboflavin attenuates NOX2-dependent superoxide generation, lessens cytochrome c release, and inhibits apoptosome activation
The entire clinical development program, every pharmacokinetic study, every efficacy trial, every safety assessment, was conducted using subcutaneous injection
HIF-1 triggers long-lasting glutamate excitotoxicity via system x c in cerebral ischaemia-reperfusion
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