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For instance, primary microglial cells from aged (1824-months-old) rats treated with an inflammatory stimulus [i.e., transforming growth factor- (TGF-)] creates a microglial-deposited ECM that promotes the differentiation of OPCs into astrocytes, thereby preventing oligodendrocyte differentiation and myelination (Baror et al., 2019)

Although several drugs are under investigation, there are currently no approved drugs for NAFLD ( The pathogenetic role of oxidative stress in NALFD is well known and would explain the rationale for the use of GSH as a potential therapy

Pediatr Blood Cancer 46:624629 [DOI] [PubMed] [Google Scholar] Grygiel-Grniak B (2014) Peroxisome proliferator-activated receptors and their ligands: nutritional and clinical implications-a review