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Despite the many prevalent hypotheses around the pathogenesis of the disease, most treatments focus on targeting and lowering the accumulation of ammonia as well as inflammation

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Animal, in vitro and some human studies have demonstrated that ketogenic intervention not only modulates AD putative biomarkers, such as A 42/40, p-tau, GFAP and NFL, but also plays an effective role in the improvement of oxidative stress, inflammation, and mitochondrial mechanism

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Im Vergleich zur Kontrollgruppe wies die Carnitin-Gruppe niedrigere Werte bei bestimmten Markern auf, die auf Muskelschden hinweisen