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GSH depletion and concomitant increase in O&NS and mitochondrial dysfunctions play a role in the pathophysiology of diverse neuroimmune disorders, including depression, myalgic encephalomyelitis/chronic fatigue syndrome and Parkinsons disease, suggesting that depleted GSH is an integral part of these diseases

These include confounding interactions between drugs and the nematodes bacterial food source and failure of drugs to be taken up into nematode tissues

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Methods Lipid peroxidation and antioxidant enzymes activity were estimated using spectrophotometric methods

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