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91 AGEs, formed through the reactions of proteins and lipids with high glucose levels, crosslink extracellular matrix (ECM) proteins, inhibit ECM degradation by matrix metalloproteinases (MMPs) and increase cardiac stiffness, resulting in diastolic dysfunction

Thirty rats were randomly assigned to six experimental groups: control, ATZ, ATZ + LC, ATZ + LC-loaded niosomes (LCLN), ATZ + QT, and ATZ + QT-loaded niosomes (QTLN)

PMID: 21188163

They also experience better muscle gains and have an easier time getting back into the gym after difficult workouts.*

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