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10.1094/CCHEM-03-16-0069-FI 12

Mechanistically, NOX2/ROS induces endothelial-derived ET-1 overexpression, which activates the ERK1/2 signaling pathway to mediate the shift of microglia activation to a pro-inflammatory M1 phenotype that triggers the release of inflammatory mediators

In addition, there have been numerous studies showing improvement with GH replacement therapy (raising IGF-1 levels) for a number of abnormalities including: BMD (from 138.1 to 279.4)