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Soluble A oligomers, pathological tau, and neuroinflammatory mediators disrupt synaptic plasticity, impair neurotransmission, and alter dendritic spine architecture long before extensive neuronal loss occurs

Amylin and hypertension: association of an amylin-G132A gene mutation and hypertension in humans and amylin-induced endothelium dysfunction in rats

Experimental findings suggest that Dihexa may interact with HGF proteins

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These mechanisms are complementary rather than competitive, potentially making combination use attractive for anti-aging focused researchers