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doi: 10.3390/nu15214699
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The Tumor Suppressor p53 Limits Ferroptosis by Blocking DPP4 Activity
After ligand binding, the activated IGF-1 or insulin receptor phosphorylates several intracellular substrates, including IR substrates (IRS) and the Src-homology-2-domain containing transforming protein (Shc)
That added component is what shifts the blend more clearly toward inflammation-related and barrier-oriented research logic, rather than a repair-only emphasis
