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Description
AnkB-deficient animals have sinus bradycardia, a prolonged QT interval, and other myocardial electrical phenotype changes, promoting myocardial mechanical remodelling as well as premature death, which is related to the low expression of -catenin in the intercalated disc and high expression in the cytoplasm caused by the loss of AnkB
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Opioid receptors (mu, delta, kappa) are densely distributed both presynaptically on primary afferent terminals in spinal laminae IV and postsynaptically on secondorder neurons, as well as throughout critical supraspinal sites including the PAG, rostral ventral medulla (RVM), locus coeruleus, and various limbic regions

Oxidative stress in the hippocampus during experimental seizures can be ameliorated with the antioxidant ascorbic acid
Oxidative stress in sickle cell disease: an overview of erythrocyte redox metabolism and current antioxidant therapeutic strategies
Improve uneven skin tone
