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Kraus, T

They can increase endothelial nitric oxide synthase (eNOS) transcription and the availability of tetrahydrobiopterin (BH4) and eNOS expression through stimulation of protein kinase B (Akt), inhibiting endothelial senescence induced by OS [71,72]

Moreover, Bach1 inactivation can induce HO-1 expression without NRF2 nuclear accumulation 53

In addition, MOS contains beta-glucans that support normal immune modulation and gut barrier function

Numerous studies revealed an association between oxidative stress and brain tumor development [11, 16]
