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This neuronal loss arises from a complex interplay of genetic predisposition and environmental exposures, which ultimately converge on a common set of molecular disturbances

Even low levels of ozone exposure can trigger the release of inflammatory and cytotoxic mediators, including myeloperoxidase (MPO), eosinophil cationic protein, lactate dehydrogenase, and albumin, all of which collectively raise ROS in the lungs and other tissues due to the oxygen-rich environment

Pang, S., et al., SKN-1 and Nrf2 couples proline catabolism with lipid metabolism during nutrient deprivation

doi:10.1017/neu.2017.2 87 Porcu, M

NO produced by iNOS nitrosates Zn-containing intracellular proteins, such as MT