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This means that glutathione can act as a reservoir of glutamate, and changes in glutathione levels can affect glutamate signaling

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Studies have also shown that the use of the autophagy inducer rapamycin can enhance autophagy by inhibiting mTOR, thereby suppressing the testosterone-induced proliferation of prostate tissue in rats [23]

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10.1142/s0192415x13500924 53 YuanJ.YanknerB

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Nevertheless, the inactivation of the proteasome leads to the failure in the elimination of oxidative mitochondrial proteins in a timely manner, then aggravating the toxicity of the damaged mitochondria and leading to more production and accumulation of ROS (104)

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[DOI] [PubMed] [Google Scholar] 48.Lykkesfeldt J

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