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This means that glutathione can act as a reservoir of glutamate, and changes in glutathione levels can affect glutamate signaling

Studies have also shown that the use of the autophagy inducer rapamycin can enhance autophagy by inhibiting mTOR, thereby suppressing the testosterone-induced proliferation of prostate tissue in rats [23]

10.1142/s0192415x13500924 53 YuanJ.YanknerB
Nevertheless, the inactivation of the proteasome leads to the failure in the elimination of oxidative mitochondrial proteins in a timely manner, then aggravating the toxicity of the damaged mitochondria and leading to more production and accumulation of ROS (104)

[DOI] [PubMed] [Google Scholar] 48.Lykkesfeldt J
