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Diabetic kidney diseases revisited: a new perspective for a new era

This could in part be due to the lower levels of ApoE in the CNS of ApoE4 carriers (Flowers and Rebeck, 2020), thus offering less overall antimicrobial protection, but also mechanistically as ApoE4 prevents HSV-1 cellular attachment but crucially not viral entry or replication, and can also be incorporated into the HSV-1 virion that can enhance release from the cell (Feng et al., 2023), which would be an issue for viral AD

Caspase-dependent cell death-associated release of nucleosome and damage-associated molecular patterns

Interference at any stage of this sequential reaction can have a profound effect on downstream signaling and metabolism