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[DOI] [PMC free article] [PubMed] [Google Scholar] Arnold SE, Arvanitakis Z, Macauley-Rambach SL, Koenig AM, Wang HY, Ahima RS, Craft S, Gandy S, Buettner C, Stoeckel LE, Holtzman DM, Nathan DM

doi: 10.1089/jmf.2009.0084

Metabolic consequences of intrauterine growth retardation

The ketone bodies produced and released by glycolytic fat cells are the ideal fuel for ATP production and they can be burned more efficiently than other mitochondrial substrates, even during hypoxia, potentially allowing the tumor grow when without adequate blood supply (279)

In addition, various cytostatic drugs induced neutropenia in patients as well as in experimental animals
