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In line with a previous study by the authors, the rifaximin-mediated blunting of intestinal hyperpermeability suppressed the CDAA-induced hepatic proinflammatory response and fibrogenesis by inhibiting hepatic the LPS/TLR4 signaling pathway (23)
Studies show that a shift from glucose to lipid metabolism occurs in PD and is clearly associated with insulin resistance 5,6,7,9,91
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