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doi: 10.1016/j.annemergmed.2004.08.040

Brett King, a Yale dermatologist who was the principal investigator for the two trials

Liver samples were filtered using a 10 kDa filter

As demonstrated in the experiment conducted by Khan et al

Notably, breast cancer cells achieve metabolic adaptation by hijacking the autophagy mechanism: under glucose-deprivation conditions, MANF-mediated mitophagy eliminates dysfunctional mitochondria through the PRKN/Parkin-dependent pathway, maintains the ATP supply and promotes tumour survival [112], suggesting that targeting the MANFPRKN interaction can break the drug resistance barrier of breast cancer under metabolic stress