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2000spresent Advances in animal models and immunogenetics reveal Crohn disease as a dysregulated immune response to commensal microbiota, with clinical subtypes (inflammatory, stenotic, fistulizing) echoing those described in 1932

This approach is particularly useful for refractory dermatophyte infections or mixed fungal-inflammatory presentations where a single commercial antifungal has proven insufficient

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A formative time in our understanding of formate
