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Kalekar, L

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Additionally, it may regulate immune responses, reducing the risk of autoimmune conditions and inflammatory disorders

Furthermore, in a study using autologous recipients, one could not determine whether protective compensatory mechanisms in the plasma of G6PD-deficient subjects could compensate for their poor redox status, as previously reported (55), or whether the difference in PTR is due to an intrinsic defect of G6PD-deficient RBCs as opposed to extrinsic effects of G6PD-deficiency on RBC clearance