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Specifically, iron overload conditions have been shown to increase the expression and activity of T-type calcium channels, leading to enhanced iron influx into the cardiomyocytes

ACSL4-dependent ferroptosis does not represent a tumor-suppressive mechanism but ACSL4 rather promotes liver cancer progression

If injected in high doses, it can put stress on the kidneys or lead to toxic buildup in the body, leading to serious health concerns and even death

Figure 6 demonstrates the molecular mechanisms involved in the effects of nanoparticles on COPD and pulmonary fibrosis (PF)

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