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The peptide's upregulation of VEGFR2 at both mRNA and protein levels sensitizes healing tissues to angiogenic signals while its activation of the Src-Caveolin-1-eNOS pathway generates nitric oxide essential for vascular development and tissue perfusion

Combination-program pharmacology

First identified as a potential neuroprotective agent, in 2008, by researchers in Germany, [20] 9-me-BC was initially found to be of interest due to its ability to upregulate tyrosine hydroxylase, the enzyme responsible for converting the amino acid L-theanine into the dopamine precursor, L-DOPA

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