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However, the energy needed to sustain cardiomyocyte physiological activity is supplied by both glucose and FA metabolism, although FA oxidation rather than glucose metabolism is the predominant energy source ( The metabolic link between FA and cardiomyocyte injury induced by a large number of risk factors has attracted much attention
This indicates that apoC1 in macrophages is not likely to play a major role in the development of atherosclerotic plaques in vivo, despite its promoting effect on cholesterol efflux demonstrated in vitro

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As a result, CT2 may be a new candidate for regulating the doxorubicin influx and increasing intracellular doxorubicin accumulation
