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Direct antifibrotic therapeutic actions may become more critical when liver disease is already too advanced to allow sufficient time for primarily causal therapeutics to act, since clinical progression of advanced fibrosis may occur more rapidly than previously anticipated.24 Both cholestatic and fatty liver diseases are characterised by complex cellular interaction of hepatocytes with cholangiocytes, HSCs and proinflammatory cells, such as monocyte-derived macrophages, resident Kupffer cells and lymphocytes

The above studies indicated that BaP induced lung carcinogenesis by altering methylation of genes

Anesthetics influence leukocyte adhesion, migration, activation and the release of inflammatory mediators

10.3389/fimmu.2021.628741 247 KrishnanM.MydeenA