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Studies on the cellular and molecular pathways of autophagy showed that acute exposure to oxidative stress increased autophagy activity, while chronic exposure to oxidative stress reduced autophagy

NO-agents, L-NAME and/or L-arginine have diverse effects, the initial weakening of blood vessel disappearance and finally, severe worsening of adhesions (L-NAME) vs the initial weakening of blood vessel disappearance and finally, attenuation of adhesions formation (L-arginine), which counteract each other response given together

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