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Studies in human TfR knockin (TfRmu/hu KI) mice and nonhuman primates (NHPs) confirmed the broad distribution of therapeutic ASOs across brain regions, highlighting the OTVs potential for treating AD [121]
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Predictably, their objective inflammatory markers spiked, and their symptoms relapsed, establishing a strong causal relationship between the environmental trigger and the biological response
GSH depletion in rats affects mitochondrial function during liver regeneration, resulting in diminished liver regeneration [40]
The wild type flies, irrespective of their treated conditions showed strong negative geotaxis through a vertical column
