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65th Annual Southern Thoracic Surgical Association, 2018

Among the identified sites, H3K18la is the best characterized, promoting the transcription of tissue-repair and anti-inflammatory genes such as Arg1 during M1-to-M2 macrophage polarization [15, 84]

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This discrepancy implies that additional layers of regulation, including altered translational efficiency and/or increased protein turnover, probably further contribute to cholesterolinduced PPAR downregulation, representing compelling areas for future investigation