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Researchers propose that the peptide's brief presence is sufficient to trigger a cascade of gene expression changesincluding activation of transcription factors c-Fos, c-Jun, and Egr-1that initiate self-sustaining healing processes well beyond the peptide's physical presence
The stress protein TP53INP1 plays a tumor suppressive role by regulating metabolic homeostasis
Importantly, M2 TAMs secrete chemokines such as CCL22 and CCL18, which recruit Tregs and further reinforce an immunosuppressive loop [41]
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