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In contrast, p53 R175H, L194F, R249S, and R280K mutants lose the ability to activate the Bax/Bak lipid pore and alter VDAC multimerization state, which inhabit apoptosis in cancer cells [105]

Kupffer cells and hepatic stellate cells (HSCs) can be activated by HCV-related proteins 78,79,80,81,82,83

Wild-type p53 (wtp53) plays pivotal role in many important biological processes by regulating the transcription of several target genes [22]

Capsaicin protects against testicular torsion injury through mTOR-dependent mechanism