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P.MiolaM.LuccaT.et al (1997)

Uno de los mecanismos clave en la fisiopatologa de la obesidad es la inflamacin crnica de bajo grado, mediada por la produccin de citocinas proinflamatorias y adipocinas que contribuyen al desarrollo de resistencia a la insulina y a una activacin persistente del sistema nervioso simptico, lo que genera un aumento de la presin arterial y favorece la disfuncin endotelial, incrementando el riesgo cardiovascular

S., Amaro, A., et al
