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From a pathophysiological perspective, my first thought is that this could reflect an indirect effect on peripheral nerve sensitivity, possibly related to: rapid shifts in glucoselipid metabolism, changes in nerve conduction due to electrolyte fluctuations, or even a centrally mediated effect, considering GLP-1 receptor expression in the CNS

Hypoglycemia is a serious potential side effect for anyone on a GLP-1

GLP-1 medications address several of these pathways simultaneously, and stopping treatment may allow the underlying PCOS-driven metabolic dysfunction to reassert itself