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Recent clinical trials demonstrating the efficacy of newer GLP-1 medicines across a broader range of diseases raise a fundamental question in GLP-1 pharmacology: Is metabolic control alone sufficient to explain such broad efficacy

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The engineering does not allow it

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In -cells, TMACs catalyze conversion of ATP to cytosolic cAMP, a second messenger that activates either protein kinase A (PKA) or the cAMP-regulated guanine nucleotide exchange factor designated as Epac2