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It is suggested that an overproduction of A sparks peptide self-aggregation into soluble oligomers and rigid fibrillar arrays, ultimately giving rise to the amyloid deposits seen in pathological specimens (Soria Lopez et al., 2019)

Khandaker GM, Zimbron J, Dalman C, Lewis G, Jones PB

Values are means standard deviations

Georgiadou SP, Sampsonas FL, Rice D, Granger JM, Swisher S , Kontoyiannis DP

Observations that rats treated with CsA develop hypertension prior to myocardial hypertrophy (4, 101, 153155) supported the notion that perhaps the clinical hypertrophic phenotype is purely related to CNI-induced hypertension rather than direct myocardial effects